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A dedicated outcomes RCT is needed to confirm whether these complementary effects translate into complementary and non-redundant reductions in hard endpoints
This is by design: the short duration prevents the sustained GH elevation that would trigger negative feedback and somatostatin rebound
As shown in Table 2
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Role of oxidant stress in endothelial dysfunction produced by experimental hyperhomocyst(e)inemia in humans
Ferreira et al., 2021)
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